In a recent study published in Cells, Dr. Andrea Fuso explores the potential of Vitamin K2, particularly menaquinone-7, in supporting neurological health. Through experimentation on neuroblastoma cells, the study reveals that K2 supplementation may mitigate neurodegeneration and neuroinflammation by modulating gene expression.
Specifically, Vitamin K2 reduces the expression of genes associated with neurodegeneration and neuroinflammation while promoting genes that protect against amyloid formation. Moreover, the study uncovers a correlation between DNA methylation patterns and the regulation of certain genes linked to neurological health.
This research adds to the growing body of evidence regarding the impact of natural compounds on brain function. Dr. Fuso’s prior work on S-Adenosyl-Methionine (SAMe) highlighted its role in cognitive decline, emphasizing the importance of methyl-donor availability in healthy aging. Similarly, Vitamin K2 emerges as a promising candidate for cognitive support, offering another avenue for understanding the intricate relationship between nutrition and epigenetic modulation of brain diseases.
Connecting this research with Adonat® Premium SAMe, underscores the significance of both molecules in maintaining cognitive health. While SAMe influences gene activity and memory enhancement, Vitamin K2 shows potential in mitigating neurodegeneration and neuroinflammation. Together, these findings suggest a synergistic approach to cognitive support, where both Adonat® and MenaQ7® could play complementary roles in promoting brain health and cognitive function.
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